ISBN: 9783540763307
During most of the 20th century, neurodegenerative diseases remained among the most enigmatic disorders of medicine. The scientific study of these conditions was descriptive in nature, de… Mehr…
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ISBN: 9783540763307
During most of the 20th century, neurodegenerative diseases remained among the most enigmatic disorders of medicine. The scientific study of these conditions was descriptive in nature, de… Mehr…
hive.co.uk No. 9783540763307. Versandkosten:Instock, Despatched same working day before 3pm, zzgl. Versandkosten. Details... |
ISBN: 9783540763307
During most of the 20th century, neurodegenerative diseases remained among the most enigmatic disorders of medicine. The scientific study of these conditions was descriptive in nature, de… Mehr…
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2008, ISBN: 9783540763307
eBooks, eBook Download (PDF), 2008, [PU: Springer Berlin], Seiten: 183, Springer Berlin, 2008
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2008, ISBN: 9783540763307
eBooks, eBook Download (PDF), 2008, [PU: Springer Berlin], Springer Berlin, 2008
lehmanns.de Versandkosten:Download sofort lieferbar. (EUR 0.00) Details... |
ISBN: 9783540763307
During most of the 20th century, neurodegenerative diseases remained among the most enigmatic disorders of medicine. The scientific study of these conditions was descriptive in nature, de… Mehr…
ISBN: 9783540763307
During most of the 20th century, neurodegenerative diseases remained among the most enigmatic disorders of medicine. The scientific study of these conditions was descriptive in nature, de… Mehr…
ISBN: 9783540763307
During most of the 20th century, neurodegenerative diseases remained among the most enigmatic disorders of medicine. The scientific study of these conditions was descriptive in nature, de… Mehr…
2008, ISBN: 9783540763307
eBooks, eBook Download (PDF), 2008, [PU: Springer Berlin], Seiten: 183, Springer Berlin, 2008
2008, ISBN: 9783540763307
eBooks, eBook Download (PDF), 2008, [PU: Springer Berlin], Springer Berlin, 2008
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Detailangaben zum Buch - Synaptic Plasticity and the Mechanism of Alzheimer's Disease
EAN (ISBN-13): 9783540763307
ISBN (ISBN-10): 3540763309
Erscheinungsjahr: 2008
Herausgeber: Springer Berlin
183 Seiten
Sprache: eng/Englisch
Buch in der Datenbank seit 2011-08-29T10:12:32+02:00 (Berlin)
Detailseite zuletzt geändert am 2023-11-16T23:46:28+01:00 (Berlin)
ISBN/EAN: 3540763309
ISBN - alternative Schreibweisen:
3-540-76330-9, 978-3-540-76330-7
Alternative Schreibweisen und verwandte Suchbegriffe:
Autor des Buches: triller, yves christe, philipp springer, schlüter
Titel des Buches: plasticity, alzheimer disease
Daten vom Verlag:
Autor/in: Dennis J. Selkoe; Antoine Triller
Titel: Research and Perspectives in Alzheimer's Disease; Synaptic Plasticity and the Mechanism of Alzheimer's Disease
Verlag: Springer; Springer Berlin
183 Seiten
Erscheinungsjahr: 2008-01-08
Berlin; Heidelberg; DE
Sprache: Englisch
149,79 € (DE)
154,00 € (AT)
177,00 CHF (CH)
Available
XII, 183 p. 34 illus., 26 illus. in color.
EA; E107; eBook; Nonbooks, PBS / Medizin/Nichtklinische Fächer; Neurowissenschaften; Verstehen; Alzheimer; Parkinson; alzheimer's disease; amyloid precursor protein; attention deficit disorder (ADD); autism; autism spectrum disorder; brain; cortex; glutamate receptor; memory; postsynaptic density; receptor; receptor trafficking; synapse loss; B; Neuroscience; Human Physiology; Biomedical and Life Sciences; Physiologie; BB
Permanence of the Synapse and Molecular Instability.- Cellular Biology of AMPA Receptor Trafficking and Synaptic Plasticity.- Imaging of Experience-Dependent Structural Plasticity in the Mouse Neocortex in vivo.- Synapse Loss, Synaptic Plasticity and the Postsynaptic Density.- Impact of Beta Amyloid on Excitatory Synaptic Transmission and Plasticity.- Quantitative Neuropathology in Alzheimer’s Mouse Models.- Multiple Levels of Synaptic Regulation by NMDA-type Glutamate Receptor in Normal and Disease States.- Soluble Oligomers of the Amyloid ?-Protein: Impair Synaptic Plasticity and Behavior.- Why Alzheimer’s is a Disease of Memory: Synaptic Targeting by Pathogenic A? Oligomers (ADDLs).- Synaptic Transmission Dynamically Modulates Interstitial Fluid Amyloid-? Levels.- A?-Induced Toxicity Mediated by Caspase Cleavage of the Amyloid Precursor Protein (APP).- Long-Term Potentiation and A?: Targeting A? Species, Cellular Mechanisms and Putative Receptors.- Genes, Synapses and Autism Spectrum Disorders.Includes supplementary material: sn.pub/extras
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